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產(chǎn)品詳情
  • 產(chǎn)品名稱(chēng):Anti-FAM105A抗體

  • 產(chǎn)品型號(hào):FAM105A蛋白抗體
  • 產(chǎn)品廠商:KALANG
  • 產(chǎn)品文檔:
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Anti-FAM105A抗體產(chǎn)品質(zhì)量穩(wěn)定,實(shí)驗(yàn)效果明顯,貨期快,價(jià)格優(yōu)惠,歡迎垂詢(xún)訂購(gòu)!我公司長(zhǎng)期供應(yīng)**組化抗體、WB抗體、**組化試劑盒和抗體試驗(yàn)所需全部相關(guān)試劑、熒光標(biāo)記抗體、單克隆抗體、多克隆抗體、各種標(biāo)記的二抗IgG/IgM/IgD/IgA等科研實(shí)驗(yàn)抗體。Anti-FAM105A抗體用于**組化實(shí)驗(yàn),WB實(shí)驗(yàn),相應(yīng)的標(biāo)記抗體有HRP標(biāo)記抗體,FITC標(biāo)記,BIO等。
詳情介紹:
Rabbit  Anti-FAM105A
Cat. Number:
Anti-FAM105A抗體KL-14688R
Quantity size:
0.2ml
Concentration:
1mg/ml   Buffer = 0.01M TBS(pH7.4) with 1% BSA, 0.03% Proclin300 and 50% Glycerol.
Background:
With 181 million base pairs encoding around 1,000 genes, chromosome 5 is about 6% of human genomic DNA. It is associated with Anti-FAM105A抗體Cockayne syndrome through the ERCC8 gene and familial adenomatous polyposis through the adenomatous polyposis coli (APC) tumor suppressor gene. Treacher Collins syndrome is also chromosome 5 associated and is caused by insertions or deletions within the TCOF1 gene. Deletion of the p arm of chromosome 5 leads to Cri du chat syndrome. Deletion of 5q or chromosome 5 altogether is common in therapy-related acute myelogenous leukemias and myelodysplastic syndrome. The FAM105A gene product has been provisionally designated FAM105A pending further characterization.
Also known as:
F105A_HUMAN; Fam105a; Family Anti-FAM105A抗體with sequence similarity 105 member A; FLJ11127; FLJ43660; Hypothetical protein LOC54491; Protein FAM105A.
Specificity:
Rabbit Polyclonal IgG, affinity purified by Protein A.
Reacts with: Human, Mouse, Rat, Anti-FAM105A抗體Dog, Cow, Sheep, .
Immunogen: KLH conjugated synthetic peptide derived from human FAM105A.
Predicted Molecular Weight: 42kDa.
Storage:
Shipped at 4℃, Store at -20℃ (Avoid repeated freeze/thaw cycles).
Application:
WB=1:100-500  ELISA=1:500-1000  IHC-P=1:100-500  IHC-F=1:100-500  ICC=1:100-500  IF=1:100-500   
Not yet tested in other applications. Anti-FAM105A抗體
Optimal working dilutions must be determined by the end user.
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